infarction

Definitions

  • WordNet 3.6
    • n infarction localized necrosis resulting from obstruction of the blood supply
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Webster's Revised Unabridged Dictionary
    • Infarction An infarct{2}; as, a cardiac infarction .
    • Infarction (Med) The act of stuffing or filling;
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Century Dictionary and Cyclopedia
    • n infarction The act of stuffing or filling; the condition of being stuffed; the substance with which something is stuffed or filled. Formerly applied in pathology to a variety of morbid local conditions; now usually restricted to certain conditions caused by a local fault in the circulation.
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Etymology

Webster's Revised Unabridged Dictionary
See Infarce

Usage

In literature:

I believe it to be caused by an infarction of the lungs.
"An Account of the Foxglove and some of its Medical Uses" by William Withering
This illustrates what are called infarctions.
"Special Report on Diseases of Cattle" by U.S. Department of Agriculture
Anemic infarcts may not occur.
"Arteriosclerosis and Hypertension:" by Louis Marshall Warfield
The engorgement of the obstructed territory has received the name of hemorrhagic infarction.
"A System of Practical Medicine by American Authors, Vol. I" by Various
There may be pulmonary infarcts.
"Scurvy Past and Present" by Alfred Fabian Hess
Perhaps it was an infarction.
"My Experiences in a Lunatic Asylum" by Herman Charles Merivale
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In news:

As a result, "there are heart failure patients and patients with acute myocardial infarction that are paying the price.".
For years researchers have experimented with stem cells in attempts to regrow myocardium in patients who suffer permanent tissue damage from myocardial infarction .
The role of magnesium in the regulation of various processes in cellular metabolism suggests that it may have a beneficial effect in patients with myocardial infarction (MI).
Secondary trial outcomes will measure the benefit of RIC in reduction of infarct size at 30 days and 6 months, and on 6 month HHF.
A recent study conducted at the University of Amsterdam has found that cerebral infarction is a serious concern in adults who contract community-acquired bacterial meningitis.
BioLineRx Announces Initiation of CE Mark Registration Trial for BCM (BL-1040), a Novel Medical Device for Prevention of Cardiac Remodeling Following Acute Myocardial Infarction .
New Guidelines Issued for Diagnosis and Management of Post–Myocardial Infarction Depression.
COMMIT (ClOpidogrel and Metoprolol in Myocardial Infarction Trial) collaborative group.
Despite improvements in the emergency treatment of myocardial infarction (MI), early mortality and morbidity remain high.
ACC Certifies Software Vendor for its Acute Myocardial Infarction Registry.
New Guidelines Issued for Diagnosis and Management of Post– Myocardial Infarction Depression.
Membranous nephropathy with nephrotic syndrome occurred in a patient with anterior myocardial infarction 2 months after the start of clopidogrel treatment.
Myocardial Infarction Rule-out in the Emergency Department: Are High-Sensitivity Troponins the Answer.
This 86-year-old female presented with inferior ST-elevation myocardial infarction.
Checking for both renal insufficiency and diabetes can help predict which acute myocardial infarction (AMI) patients will have cardiovascular complications and potentially die, according to this large prospective study.
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In science:

Postischemic treatment with nimesulide markedly reduced the increase in PGE2 levels in the ischemic cerebral cortex 24 h after stroke and diminished infarct size by 48 % with respect to vehicle-treated animals after 3 days of reperfusion.
Post-ischaemic treatment with the cyclooxygenase-2 inhibitor nimesulide reduces blood-brain barrier disruption and leukocyte infiltration following transient focal cerebral ischaemia in rats
Although a significant amount of ischemic tissue dies in the core of the infarct within few hours after the vessel occlusion, there is evidence showing that the damage in the surrounding tissue (ischemic penumbra) progresses over a relative long period of time (Iadecola and Ross 1997;Dirnagl et al. 1999).
Post-ischaemic treatment with the cyclooxygenase-2 inhibitor nimesulide reduces blood-brain barrier disruption and leukocyte infiltration following transient focal cerebral ischaemia in rats
An investigator blinded to the animal treatment performed the measurements of infarct volume by manually outlining the margins of infarcted areas.
Post-ischaemic treatment with the cyclooxygenase-2 inhibitor nimesulide reduces blood-brain barrier disruption and leukocyte infiltration following transient focal cerebral ischaemia in rats
The unstained area of the fixed brain section was defined as infarcted.
Post-ischaemic treatment with the cyclooxygenase-2 inhibitor nimesulide reduces blood-brain barrier disruption and leukocyte infiltration following transient focal cerebral ischaemia in rats
Infarct volume, PGE2 data, MPO activity, EB content and percent of edema were analyzed using t-test (2 groups) or one-way ANOVA with post-hoc Student-Newman-Keuls test (multiple comparison).
Post-ischaemic treatment with the cyclooxygenase-2 inhibitor nimesulide reduces blood-brain barrier disruption and leukocyte infiltration following transient focal cerebral ischaemia in rats
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