cerebral cortex

Definitions

  • WordNet 3.6
    • n cerebral cortex the layer of unmyelinated neurons (the grey matter) forming the cortex of the cerebrum
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Usage

In literature:

This, of course, is equivalent to postulating the cerebral cortex as the exclusive seat of higher intellection.
"A History of Science, Volume 4(of 5)" by Henry Smith Williams
This woman was wearing one of Ann's patented grids, sealed into her cerebral cortex.
"The Cartels Jungle" by Irving E. Cox, Jr.
Detached from the control panel of his cerebral cortex his body mechanism functioned perfectly.
"The Instant of Now" by Irving E. Cox, Jr.
Beyond they disperse again over the cortex mantle to all the centers of coordination, higher cerebration and higher psychic activities.
"The Brain" by Alexander Blade
It is therefore to items regarding the functions of the great sheet of cerebral cortex that we may now pass.
"Encyclopaedia Britannica, 11th Edition, Volume 4, Slice 4" by Various
All characteristic features of the full epileptic onset can be produced in animals deprived of the related cerebral cortex.
"Degeneracy" by Eugene S. Talbot
The main seat of this principle of life is in the central nervous system and especially in the cerebral cortex.
"Psychotherapy" by James J. Walsh
A and B, Ganglion cells from the cerebral cortex; in A the only slightly branched axon may extend the whole length of the spinal cord.
"Encyclopaedia Britannica, 11th Edition, Volume 7, Slice 8" by Various
As we have already seen, the cerebral cortex is the seat of the chief mental faculties of man or at least of the highest of these.
"Being Well-Born" by Michael F. Guyer
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In news:

IBM computer simulates cat's cerebral cortex .
On present evidence, the only tissues that last a lifetime seem to be the neurons of the cerebral cortex, the inner lens cells of the eyes, and a woman's oocytes (eggs).
In the autumn of 1906, Alois Alzheimer presented his now-famous lecture "On a Peculiar, Severe Disease Process of the Cerebral Cortex" to the 37th Assembly of Southwest German Alienists in Tübingen, Germany.
Steve Jobs, Cerebral Cortex and Unrequited Love.
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In science:

We propose that some implementation of those feed back loops is wide spread in the cerebral cortex.
A model of memory, learning and recognition
Regional differences in the synaptogensis in human cerebral cortex.
Interplay between Network Topology and Dynamics in Neural Systems
As noticed quite early in the historical debate, the cut may even be placed deep into the human observer, whose consciousness, which may be located in the cerebral cortex, represents the final link in the observational chain.
Decoherence: Basic Concepts and Their Interpretation
Unlike the cerebral cortex, in the subcortical areas, this initial EB leakage persisted till 6 h, showing a significant increase in EB extravasation as compared to the contralateral side (Fig. 2B).
Post-ischaemic treatment with the cyclooxygenase-2 inhibitor nimesulide reduces blood-brain barrier disruption and leukocyte infiltration following transient focal cerebral ischaemia in rats
By measuring the activity of an enzymatic marker of these inflammatory cells (MPO), we assessed the degree of leukocyte infiltration into the cerebral cortex and subcortical regions following the focal ischemic event.
Post-ischaemic treatment with the cyclooxygenase-2 inhibitor nimesulide reduces blood-brain barrier disruption and leukocyte infiltration following transient focal cerebral ischaemia in rats
Maximal leukocyte infiltration was seen at 24-48 h in the cerebral cortex, and at 12-24 h post-reperfusion in the subcortical areas (Figs 3A and 3B). No significant changes in EB leakage, edema and MPO activity, were observed between the ipsilateral and contralateral side of rats that underwent a sham operation.
Post-ischaemic treatment with the cyclooxygenase-2 inhibitor nimesulide reduces blood-brain barrier disruption and leukocyte infiltration following transient focal cerebral ischaemia in rats
Prostaglandin E2 concentrations in the ischemic cortex are reduced by nimesulide but not by the COX-1 inhibitor VAS We investigated the effect of selective inhibition of either COX-1 or COX-2 on PGE2 levels in the ischemic cerebral cortex after 24 h of reperfusion.
Post-ischaemic treatment with the cyclooxygenase-2 inhibitor nimesulide reduces blood-brain barrier disruption and leukocyte infiltration following transient focal cerebral ischaemia in rats
Administration of the COX-2 inhibitor nimesulide produced a significant protective effect against ischemia-induced PGE2 accumulation in the cerebral cortex, keeping PGE2 concentrations at the basal level (compared to ipsilateral side of sham-operated animals).
Post-ischaemic treatment with the cyclooxygenase-2 inhibitor nimesulide reduces blood-brain barrier disruption and leukocyte infiltration following transient focal cerebral ischaemia in rats
Adams J., Collaco-Moraes Y. and de B. J. (1996) Cyclooxygenase-2 induction in cerebral cortex: an intracellular response to synaptic excitation. J.
Post-ischaemic treatment with the cyclooxygenase-2 inhibitor nimesulide reduces blood-brain barrier disruption and leukocyte infiltration following transient focal cerebral ischaemia in rats
Infarct volumes were calculated from six coronal TTC-stained brain slices, and assessed in the cerebral cortex and subcortical regions.
Post-ischaemic treatment with the cyclooxygenase-2 inhibitor nimesulide reduces blood-brain barrier disruption and leukocyte infiltration following transient focal cerebral ischaemia in rats
BBB breakdown was assessed by quantifying the concentration of Evans Blue leakage into the cerebral cortex (A) and subcortical areas (B).
Post-ischaemic treatment with the cyclooxygenase-2 inhibitor nimesulide reduces blood-brain barrier disruption and leukocyte infiltration following transient focal cerebral ischaemia in rats
Production of PGE2 in the ischemic cerebral cortex is potently reduced by the COX-2 inhibitor nimesulide, but only very modestly diminished by the highest dose of the COX-1 inhibitor valeryl salicylate (VAS).
Post-ischaemic treatment with the cyclooxygenase-2 inhibitor nimesulide reduces blood-brain barrier disruption and leukocyte infiltration following transient focal cerebral ischaemia in rats
Concentrations of EB were determined in the ischemic cerebral cortex (A) and subcortical areas (B). °p<0.001 with respect to the contralateral side; **p<0.01 and *p<0.05 with respect to the stroke side of vehicleadministered animals.
Post-ischaemic treatment with the cyclooxygenase-2 inhibitor nimesulide reduces blood-brain barrier disruption and leukocyte infiltration following transient focal cerebral ischaemia in rats
Hippocampal MnSOD activity significantly increased at 24, 48 and 72 h of reperfusion (57, 46 and 51% vs. control, respectively). MnSOD activity did not change in cerebral cortex or striatum at any time point examined.
Time course of oxidative damage in different brain regions following transient cerebral ischemia in gerbils
Adams, J., Collaço-Moraes, Y., de Belleroche, J., 1996. Cyclooxygenase-2 induction in cerebral cortex: an intracellular response to synaptic excitation. J.
Nimesulide limits kainate-induced oxidative damage in the rat hippocampus
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